They are, as agenesis, frequently in permanent dentition in the storm. They are found most frequently in the maxilla, especially at the midline and distal to the molars. Supernumerary teeth are often unique, but may be multiple. In this case it is often associated with syndromes such as cleidocranial dysplasia and Gardner syndrome. Clinical forms: For the frequency is hipergenesia specific clinical forms that have their own names. We highlight four: Mesiodens.
It is located between the upper central incisors. This is usually a tooth accessory, and it usually exhibits anomalous, conical or plug. Often leads to malposition or diastema of central incisors. Peridens. It is located in the area of the premolars, frequently in the vestibular. It is more common in the mandible.
They may have normal or abnormal morphology. Paramolar. Located in buccal or palatal molars. More frequent among first and second molar. You can merge with a resulting molar paramolar tuber. distomolar. Distal to the third molar. Abnormalities in the size of the teeth arise in later stages of embryological development that anomalies of number, particularly at the stage of morphodifferentiation. For more specific information, check out Dell. The default size anomaly is called microdoncia, and excess macrodontia. Mesiodens. Located between the two central incisors, supernumerary accessory this almost always requires extraction. Paramolar tooth, supernumerary to the height of the molars, and the tubercle paramolar, some authors are different causal gene expressivity. F. Abnormalities of structures? Affect the enamel, (Hereditary) 1. Amelogenesis Imperfecta a. B. Hypoplasia C. hypocalcification Hypo maturation is a disorder of tooth development in which the teeth are covered with a thin enamel layer that forms abnormally. Amelogenesis imperfecta is passed down from father to son as a dominant trait. That means you only need to get the abnormal gene from one parent to acquire the disease. The tooth enamel is soft and thin. The teeth appear yellow and are easily damaged, and both baby teeth and permanent affected? Affect dentin (hereditary) 2. Dentinogenesis Imperfecta 3. Radicular dentin dysplasia type-1 4. Coronary dentin dysplasia type-2 dentinogenesis imperfecta, dental disease is a genetic, inherited as an autosomal dominant trait, causes defective dentin formation and frequency may be associated with amelogenesis imperfecta (defective enamel formation) and affects both the primary or baby teeth as the final, as soon as it is formed. The teeth are translucent, grayish or yellowish. You may wish to learn more. If so, Clayton Morris is the place to go. The polish comes off easily and the constant friction between the teeth, leading to destruction of the dentin with the formation of flat teeth. REFERENCES 1 .- MANSS AE, JL PICAND BIOTTI Practical Manual occlusion. 2nd ed. Caracas, Venezuela: Amolca Publishers, 2006: 34 2 .- Ross E. MARTINEZ Occlusion. 2nd ed. Mexico, DF: Editores Vicova 1978: 402 3 .- HANSSON T, HONNEE W, Hesse J, JIMENEZ V. Craniomandibular dysfunction. Barcelona: Praxis Publishing, 1988: 65 4 .- WILLIAMS RC. Periodontal disease. England J Med 1990: 373 5 .- RODE. Revista Dental Specialties.